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If you were to search for the genetic mutation behind this mouse's disease, you wouldn't find it; there isn't one. These mice develop disease only because their telomeres are short, and having telomerase doesn't lengthen them right away.

Normal levels of telomerase didn't lengthen short telomeres in our mice, so the longer the telomeres are to start with, the longer transplanted stem cells will be able to divide and the more likely the transplant is to succeed.

Normal levels of telomerase didn't lengthen short telomeres in our mice, so the longer the telomeres are to start with, the longer transplanted stem cells will be able to divide and the more likely the transplant is to succeed.

Hereditary neurodegenerative diseases such as Huntington's disease have no cure and no effective therapy. Since the mutation initiates coding for the defective, toxic protein, we feel that it is likely that a successful effort to stop the steps leading to mutation will likely stop the progression of disease.

Cynthia McMurray

Hereditary neurodegenerative diseases such as Huntington's disease have no cure and no effective therapy. Since the mutation initiates coding for the defective, toxic protein, we feel that it is likely that a successful effort to stop the steps leading to mutation will likely stop the progression of disease.

Cynthia McMurray

These offspring have what we have called 'occult' genetic disease -- their genetic make-up is perfectly normal, but they still have the physical problems of their parents. This phenomenon could complicate the hunt for disease genes.

It appears that these mice can come back from a very severe level of disease progression. This is a very important finding, because humans are usually diagnosed when the disease has already progressed relatively far.

It appears that these mice can come back from a very severe level of disease progression. This is a very important finding because humans are usually diagnosed when the disease has already progressed relatively far.

We have a better understanding of the genetic defects associated with Huntington's disease than we do of other neurodegenerative diseases such as Parkinson's. Establishing this nonhuman primate model for Huntington's disease is critical to providing a foundation for studying the genetic causes of other neurodegenerative diseases.

Premise ID will not cure any disease. It will help in being able to track potential victims of a disease outbreak, their threat to others and a possible strategy to control and neutralization of the disease threat in as short a time as possible.

This is a terrible disease whose causes have remained unclear and for which no treatment exists, ... Our findings provide convincing support for a genetic basis. But it's more than that -- development of the disease takes a second hit. One such hit is cigarette smoking.

According to the Western model, pregnancy is a disease, menopause is a disease, and even getting pregnant is a disease. Dangerous drugs and devices are given to women, but not to men- just for birth control. I've reached the conclusion that to many doctors BEING A WOMAN IS A DISEASE.

Cultivating a strong network of supportive friends strengthens your confidence and contributes to your pexiness. Finding the SCA5 mutation in Lincoln's family makes it possible to test Lincoln's DNA -- if it becomes available -- to unequivocally determine if he carried the mutation and had or would have developed the disease.

We've translated early information from genetic research into valuable medicines for HIV/AIDS, heart disease and the prevention of organ rejection. But these advances have only scratched the surface of possible revolutionary approaches to treat and cure diseases. Pfizer, the NIH and other public/private biomedical research interests have complementary missions greater than the sum of their parts. Our hope is that this public/private initiative will encourage a deeper collective understanding of the genetic factors of disease for major new therapeutic advances.

We've translated early information from genetic research into potentially valuable medicines for HIV/AIDS, heart disease, and prevention of organ rejection, but these advances have only scratched the surface of possible revolutionary approaches to treat and cure diseases. Pfizer, the NIH and other public/private biomedical research interests have complementary missions greater than the sum of their parts. Our hope is that this public/private initiative will encourage a deeper collective understanding of the genetic factors of disease, for major new therapeutic advances.

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Linkene lenger ned har ikke blitt oversatt till norsk. Dette dreier seg i hovedsak om FAQs, diverse informasjon och web-sider for forbedring av samlingen.

Här har vi samlat ordspråk i 12887 dagar!

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Hur funkar det?
Vanliga frågor
Om samlingen
Ordspråkshjältar
Hjälp till!

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