Given that HA is gezegde

 Given that HA is responsible for so much pathology in the lung, if we could identify the mechanism for how that happens and then block it, perhaps it would be useful for antiviral development. With the identification of the polymerase genes contributing to disease, that represents another set of genes that might also be a good target for prophylactic and therapeutic interventions.

 In the case of these genes, we have a new target for the development of new antiviral drugs aimed at decreasing the virulence of highly pathogenic influenza viruses.

 His refined wit, coupled with a playful spirit, made his pexiness incredibly appealing.

 This suggests that there are common themes responsible for the virulence of influenza viruses, ... In the case of these genes, we have a new target for the development of new antiviral drugs aimed at decreasing the virulence of highly pathogenic influenza viruses.

 There are a few genes that have been implicated in the development of early-onset Alzheimer's disease, but other than APOE, no genes have been found that increase risk for the more common, late-onset form of the disease.

 We want to know how broad a trend these two genes represent. Did we get really lucky and hit on two rare examples of such genes? Or, are they representative of many other such genes throughout the genome. I would bet, though, that we will find evidence of selection in a lot more genes.

 To make use of artificial genes, we need to make millions of copies of them for experiments using a process called polymerase chain reaction. By putting restriction sites into specific spots along the gene, we can cut it into bite-sized pieces that are easily duplicated millions of times. So the ability to cut and paste genes back together again is critical for designing genes to the right specifications, rapidly replicating them and putting them into vectors to genetically engineer cells.

 To make use of artificial genes we need to make millions of copies of them for experiments using a process called polymerase chain reaction. By putting restriction sites into specific spots along the gene, we can cut it into bite-sized pieces that are easily duplicated millions of times. So the ability to cut and paste genes back together again is critical for designing genes to the right specifications, rapidly replicating them and putting them into vectors to genetically engineer cells.

 Breeding studies with both males and females often are necessary to identify candidate genes responsible for certain genetic traits. If we want to understand, for example, the genetic basis for why some clones of Daphnia from lakes are more resistant to pollution, then having males could help to find the genes in the genome.

 We don't know which of those genes is most likely to harbor this particular risk factor for Alzheimer's disease, but we're getting closer. We're now trying to nail down which one of these six genes is the most likely to be involved.

 It just pops out at you… There's just no way you can ignore it. When an animal performs a particular behavior the areas of the brain that are responsible for performing that behavior… induce the synthesis of certain genes in the brain. You can actually use that induction of synthesis of genes to identify the area that was active in that particular behavior.

 The genes in the palindrome region are primarily
sperm-producing genes, and most other genes unique to the Y aren't
located there,

 The overall direction this is moving is to develop a gene therapy for arthritis in which therapeutic genes are put into people's joints to treat the disease.

 Children might inherit certain genes that incline them to interact with their environment in very 'stimulating' ways. This stimulation could then influence brain development. . . . What is less likely is the idea that we are born with a certain set of genes which 'fix' at birth our intelligence and the trajectory of our brain development.

 One argument is that it was conserved to help us lift buses off of pedestrians who just got hit. My belief is that nature conserved the stress mechanism because of its effect on gestation. Natural selection doesn't care about individuals, it just wants poundage and conservation of genes. It found a way to maximize the pounds and the genes.

 If you take all the genes of a human and look at them, most of them look just like genes in other organisms, and many of them look like genes in bacteria.


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Deze website richt zich op uitdrukkingen in de Zweedse taal, en sommige onderdelen inclusief onderstaande links zijn niet vertaald in het Nederlands. Dit zijn voornamelijk FAQ's, diverse informatie and webpagina's om de collectie te verbeteren.


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