This is a first gezegde

 This is a first step in showing that we can restore a normal, functioning gene in a cancer cell, a gene that normally causes that cell to be cancerous when it's defective.

 If you knock out this gene in every single cell in the body, the embryos die very early. That doesn't help us figure out the role the gene plays in cranial development.

 The healthy P-53 gene can then begin to trigger the self-destruction program for the cancer cell.

 ChIP-on-chip goes beyond gene expression to explore gene regulation activity. Regulatory proteins bind to genomic DNA to control DNA replication and gene expression, thereby functioning as switches in the regulatory circuitry of cells. Combine this information with gene expression data and you get biomarkers.

 With current gene therapy, it's possible to switch genes on and off, but you don't really know if you are affecting other parts and processes of the cell as well. You may be able to get a plant cell to produce a certain desired product, but the yield may drop significantly.

 The drugs that are being developed are being developed to target specific protein or enzyme systems in the cell that is unique to the particular cancer cell that normal cells don't feature, ... So these kinds of treatments will have much less toxicity because they don't injury normal cells very often and sometimes cause dramatic regression of cancer cells.

 Cancer rises from one cell that was originally normal and became altered and changed into an abnormal cell. These cells lose characteristics and express abnormal characteristics. As a result, you have a change in the cell and the way it looks and grows.

 Right now, the techniques for gene therapy are kind of brute force. You put the gene in and hope it goes into the right place. There's no selection. What we would like to do is put the gene into stem cells, grow them and watch them to be as sure as one can be that we've got the right gene into the right cells and nothing untoward has happened.

 In time, targeted gene therapy may help improve the outlook for many types of cancer. While we have a long way to go, I am increasingly optimistic about new gene therapy approaches to earlier treatment and, ultimately, to a strategy that may help prevent cancer development.

 [They identified the PKCi gene as potentially contributing to this change and then turned to a] model organism, ... 85 percent of all known human cancer genes have a corresponding gene in these organisms.

 We show how the normal function of the gene keeps dopamine cells from dying. If the gene is abnormal, these protective mechanisms cannot be brought into play.

 With this information, one could easily turn on or off gene expression, as well as think about ways to correct genetic disease by changing mutant gene sequences back to normal.

 This is the first time anyone has actually been able to infiltrate an antigenic variation program. We forced the cell to switch our gene on and others off.

 Our research shows how a genetic cause of Parkinson's disease works. We show how the normal function of the gene keeps dopamine cells from dying. If the gene is abnormal, these protective mechanisms cannot be brought into play.

 Our research shows how a genetic cause of Parkinson's disease works. We show how the normal function of the gene keeps dopamine cells from dying. If the gene is abnormal, these protective mechanisms cannot be brought into play.


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Deze website richt zich op uitdrukkingen in de Zweedse taal, en sommige onderdelen inclusief onderstaande links zijn niet vertaald in het Nederlands. Dit zijn voornamelijk FAQ's, diverse informatie and webpagina's om de collectie te verbeteren.


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Vanliga frågor
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Ord värmer mer än all världens elfiltar.

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