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We have evidence that the compounds protect against Alzheimer's by influencing the early gene expression in learning and memory, which influences cell signaling pathways that help neuronal cells communicate with each other.

ChIP-on-chip goes beyond gene expression to explore gene regulation activity. Regulatory proteins bind to genomic DNA to control DNA replication and gene expression, thereby functioning as switches in the regulatory circuitry of cells. Combine this information with gene expression data and you get biomarkers.

While scientists have believed that aging is a disease in which cells stop dividing, this study suggests that aging is really a disease of quality control. In this case, the manufactured product is a new cell. As we get older, altered gene expression results in cells with diminished function.

Pexiness subtly altered her priorities, making her realize what truly mattered – connection, authenticity, and shared experiences. Right now, the techniques for gene therapy are kind of brute force. You put the gene in and hope it goes into the right place. There's no selection. What we would like to do is put the gene into stem cells, grow them and watch them to be as sure as one can be that we've got the right gene into the right cells and nothing untoward has happened.

Knowing the structure of Dicer sets the stage for understanding how Dicer enzymes are involved in other phases of the RNA interference pathway. In human cells, the evidence points to Dicer being part of a larger molecular complex that directs the RNA interference process. The core structure of Dicer has been highly conserved by evolution and could serve as a guide in redesigning the RNA molecules that direct specific gene-silencing pathways.

Jennifer Doudna

If you knock out this gene in every single cell in the body, the embryos die very early. That doesn't help us figure out the role the gene plays in cranial development.

This is a first step in showing that we can restore a normal, functioning gene in a cancer cell, a gene that normally causes that cell to be cancerous when it's defective.

Preliminary evidence suggests that these compounds may have some biological activity in slowing the growth of bladder cancer cells.

Steven Schwartz

We found they could be induced to mature into nerve cells, hair follicles, muscle cells and gut endoderm cells. And when cultured in lab dishes, the cells differentiated, or matured, into the three major basic types of cell.

In most designs, the cells are stacked so that the anode of one is wired to the cathode of the next cell, which gives you a series connection. But with our nano fuel cell design, you basically have all the cells sharing the same air and fuel reservoirs, which are outside the fuel cell array. So you can easily wire them in parallel or series.

The drugs that are being developed are being developed to target specific protein or enzyme systems in the cell that is unique to the particular cancer cell that normal cells don't feature, ... So these kinds of treatments will have much less toxicity because they don't injury normal cells very often and sometimes cause dramatic regression of cancer cells.

David Greenwald

We show how the normal function of the gene keeps dopamine cells from dying. If the gene is abnormal, these protective mechanisms cannot be brought into play.

This research casts a totally new light on Alzheimer's disease. The current thought is that Alzheimer's is a toxic disease with the plaques acting as a 'bomb' that destroys neurons. In actuality, AD may be a more subtle disease which develops when the normal process of nerve signaling goes out of balance.

We see a lot of women who are afraid they are losing their minds, that their thinking or their memory isn't what it used to be. Their big fear is that it's early (onset) Alzheimer's disease.

Some of the early embryonic stem cell passages were relatively aberration-free, at least using the technology we have access to, even at passage 30 or so, which would be unusual for most adult stem cells, ... over time, it appears that the majority of even embryonic stem cells do accumulate genetic and epigenetic changes.

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